By stimulating cancer cells to produce a molecule that activates a signaling pathway in nearby immune cells, MIT researchers have found a way to force tumors to trigger their own destruction.
Based on strong preclinical data on HMPL-A251 in HER2-positive tumor cell lines, the firm wants to take the therapy into clinical trials by year end.
Scientists from Duke-NUS Medical School and their collaborators have created one of the most comprehensive single cell maps ...
At the Hairy Cell Leukemia Foundation 2025 Conference in Bologna, Italy, we had the opportunity to sit down with Dr. Dima El-Sharkawi for a live interview. The discussion focused on how hairy cell ...
This important study examines the potential role of ARHGAP36 transcriptional regulation by FOXC1 in controlling sonic hedgehog signaling in human neuroblastoma. While there are many solid findings ...
“Hydrogen and Fuel Cells – Pathways to Progress,” reflects the growing domestic and international acceptance of H2 and FC's as alternative energy solutions A key proponent of fuel cells with ambitious ...
- Orelabrutinib, a highly selective CNS-penetrant, oral small molecule Bruton’s Tyrosine Kinase (BTK) inhibitor with best-in-class potential now in Phase 3 development for progressive forms of ...
Top panels: Root epidermal cells expressing Venus-VAMP727 and mRFP-VAMP721 in wild type (left) and the snx1 mutant (right). The punctate localization pattern of VAMP721, which functions in the ...
A new study in fruit flies describes how an animal's gut reacts differently to beneficial microbes versus harmful pathogens. A pathway known for repairing damaged genetic material, called the ...
In an FCEV, it’s all about a hydrogen fuel cell membrane. You feed it H2. It meets oxygen, magic happens—electricity for the motor, and the only tailpipe output is plain old water vapor. This is ...
Cells have a variety of processes and pathways that can repair damage, remove trash and debris, and boost healing. Scientists have now discovered a novel process in which cells eliminate stuff, and ...
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